[1] |
De Leo, Musacchio MC, Cappelli V, et al. Genetic, hormonal and metabolic aspects of PCOS: an update[J]. Reprod Biol Endocrinol, 2016, 14(1): 38.
|
[2] |
曹泽毅,主编. 中华妇产科学. 3版[M]. 北京:人民卫生出版社,2014: 2632-2666.
|
[3] |
Çelik E, Türkçüoĝlu I, Ata B, et al. Metabolic and carbohydrate characteristics of different phenotypes of polycystic ovary syndrome[J]. J Turk Ger Gynecol Assoc, 2016, 17(4): 201-208.
|
[4] |
俞健梅,马艳萍,李永刚,等. 环境内分泌干扰物对生殖健康影响的研究进展[J]. 现代生物医学进展,2014, 14(31): 6197-6200.
|
[5] |
Huang RP, Liu ZH, Yuan SF, et al. Worldwide human daily intakes of bisphenol A (BPA) estimated from global urinary concentration data (2000-2016) and its risk analysis[J]. Environ Pollut, 2017, 230: 143-152.
|
[6] |
Rochester JR. Bisphenol A and human health: a review of the literature[J]. Reprod Toxicol, 2013, 42: 132-155.
|
[7] |
Zhou W, Fang F, Zhu W, et al. Bisphenol A and ovarian reserve among infertile women with polycystic ovarian syndrome[J]. Int J Environ Res Public Health, 2016, 14(1): 18.
|
[8] |
Santangeli S, Maradonna F, Olivotto I, et al. Effects of BPA on female reproductive function: the involvement of epigenetic mechanism[J]. Gen Comp Endocrinol, 2017, 245: 122-126.
|
[9] |
Huo X, Chen D, He Y, et al. Bisphenol-A and female infertility: a possible role of gene-environment interactions[J]. Int J Environ Res Public Health, 2015, 12(9): 11101-11116.
|
[10] |
Akln L, Kendirci M, Narin F, et al. The endocrine disruptor bisphenol A may play a role in the aetiopathogenesis of polycystic ovary syndrome in adolescent girls[J]. Acta Paediatr, 2015, 104(4): e171-e177.
|
[11] |
Vahedi M, Saeedi A, Poorbaghi SL, et al. Metabolic and endocrine effects of bisphenol A exposure in market seller women with polycystic ovary syndrome[J]. Environ Sci Pollut Res Int, 2016, 23(23): 23546-23550.
|
[12] |
Kandaraki E, Chatzigeorgiou A, Livadas S, et al. Endocrine disruptors and polycystic ovary syndrome (PCOS): elevated serum levels of bisphenol A in women with PCOS[J]. J Clin Endocrinol Metab, 2011, 96(3): E480-E484.
|
[13] |
方健叶,杨菁,郭悦,等. 双酚A与多囊卵巢综合征相关性的研究进展[J]. 生殖医学杂志,2014, 23(1): 73-77.
|
[14] |
Peretz J, Craig ZR, Flaws JA. Bisphenol A inhibits follicle growth and induces atresia in cultured mouse antral follicles independently of the genomic estrogenic pathway[J]. Biol Reprod, 2012, 87(3): 63.
|
[15] |
Vagi SJ, Azziz-Baumgartner E, Sjödin A, et al. Exploring the potential association between brominated diphenyl ethers, polychlorinated biphenyls, organochlorine pesticides, perfluorinated compounds, phthalates, and bisphenol A in polycystic ovary syndrome: a case-control study[J]. BMC Endocr Disord, 2014, 14: 86.
|
[16] |
王缘,朱琴玲,何亚琼,等. 卵泡液中双酚A在多囊卵巢综合征发病中的作用及相关因素探讨[J]. 生殖与避孕,2016, 36(7): 552-556.
|
[17] |
Fernández M, Bourguignon N, Lux-Lantos V, et al. Neonatal exposure to bisphenol a and reproductive and endocrine alterations resembling the polycystic ovarian syndrome in adult rats[J]. Environ Health Perspect, 2010, 118(9): 1217-1222.
|
[18] |
周娴颖,郝树芳,谢淑武,等. 双酚A对大鼠雌性子代卵巢发育和血管内皮生长因子蛋白表达的影响[J]. 生殖与避孕,2012, 32(5): 293-300.
|
[19] |
晏佩佩,潘晓燕,王雪楠,等. 双酚A对雌性生殖器官的影响及作用机制[J]. 中国医学科学院学报,2013, 35(6): 683-688.
|
[20] |
国先芬,王贻鑫,耿存珍,等. 环境激素双酚A的毒理学研究进展[J]. 环境科学与技术,2013, 36(2): 86-92, 99.
|
[21] |
Phrakonkham P, Viengchareun S, Belloir C, et al. Dietary xenoestrogens differentially impair 3T3-L1 preadipocyte differentiation and persistently affect leptin synthesis[J]. J Steroid Biochem Mol Biol, 2008, 110(1-2): 95-103.
|
[22] |
Nadal A, Alonso-Magdalena P, Soriano S, et al. The pancreatic beta-cell as a target of estrogens and xenoestrogens: implications for blood glucose homeostasis and diabetes[J]. Mol Cell Endocrinol, 2009, 304(1-2): 63-68.
|
[23] |
Manikkam M, Tracey R, Guerrero-Bosagna C, et al. Plastic derived endocrine disruptors (BPA, DEHP and DBP) induce epigenetic transgenerational inheritance of obesity, reproductive disease and sperm epimutations[J]. PLoS One, 2013, 8(1): e55387.
|