[1] |
Schmidt NJ. An apparently new enterovirus isolated from patients with disease of the central nervous system[J]. Infect Dis, 1974, 129:304-309.
|
[2] |
Wu TN, Tsai SF, Li SF, et al. Sentinel surveillance for enterovirus 71. Taiwan, 1998[J]. Emerg Infect Dis, 1999, 5(3):458-460.
|
[3] |
Zhao SY, Li XW, Jiang ZF. Pay much attention to severe enterovirus 71 infection in children[J]. Chin J Pediatr, 2008, 46(6):40l-403.
|
[4] |
Liu YX, Xie JJ, He YX, et al. Study of clinical and laboratory features of hand-foot-mouth disease[J]. Chin J Exp Clin Virol, 2008, 22(6):475-477.
|
[5] |
Luo ST, Chiang PS, Chao AS, et al. Enterovirus 71 maternal antibodies in infants, Taiwan[J]. Emerg Infect Dis, 2009, 15(4):581-584.
|
[6] |
Chang LY, Hsiung CA, Lu CY. Status of cellular rather than humoral immunity is correlated with clinical outcome of enterovirus 71[J]. Pediatr Res, 2006, 60(4):466-471.
|
[7] |
Chen WF, ed. Medical immunology[M]. Beijing:People's Medical Publishing House, 2001, 32-33.
|
[8] |
Zhou T, Ma LZ, Fu SM, et al. Correlation analysis of hand-foot-mouth in patients with humoral immunity and its outcomes[J]. Guangdong Med J, 2010, 31(18):2434-2435.
|
[9] |
Chen DY, Huang XW. Analysis on testing serum immunoglobulins and hs-CRP in children with hand-foot-mouth disease[J]. Chin Gen Pract, 2011, 14(2b):476-477.
|
[10] |
Chang LY, Hsiung CA, Lu CY, et al. Status of cellular rather than humoral immunity is correlated with clinical outcome of enterovirus 71[J]. Pediatr Res, 2006, 60P:466-471.
|
[11] |
Maecker HT, Maino VC, Picker LJ. Immunofluorescence analysis of T-cell responses in health and disease[J]. J Clin Immunol, 2000, 20(6):391.
|
[12] |
Santana MA, Esquivel GF. Cell biology of T cell activation and differentiation[J]. Int Rev Cytol, 2006, 250:217-274.
|
[13] |
Hanna J, Mandelboim O. When killers become helpers[J]. Trends Immunol, 2007, 28(5):201-206.
|
[14] |
Wang YF, Zheng R, Chen BG, et al. Case-control study on immuneparameters and clinical significance of hand-foot-mouth disease[J]. Zhejiang Prev Med, 2010, 22(10):15-21.
|
[15] |
Qiu YR, He ZZ, Sun DH, et al. Clinical significance of T-cell subsets and double-negative regulatory T cells in hand foot mouth disease[J]. J Trop Med, 2009, 9 (1):49-51.
|
[16] |
Du BY, Bai L, Shen Y, et al. The effects of enterovirus 71 infection on the ability of T cellular immunity[J]. J Med Res, 2010, 39(8):50-53.
|
[17] |
Yang XQ. The clinical significance of the T cell subsets[J]. Chin J Pract Pediatr, 2000, 15(4):250.
|
[18] |
Chen XN. SIRS/sepsis theories and their use for guiding diagnosis and treatment of severe viral infection-A discussion based on hand, foot and mouth disease[J]. Chin Pediatr Emerg Med, 2010, 17(1):87-88.
|
[19] |
Goldstein B, Giroir B, Randolph A, et al. Internationalpediatric sepsis consensus conference: Definitions for sepsis and organ dysfunction in pediatrics[J]. Pediatr Crit Care Med, 2005, 6(1):2-8.
|
[20] |
中华医学会儿科学分会急救学组,中华医学会急诊学分会儿科组,《中华儿科杂志》编辑委员会.儿科感染性休克(脓毒性休克)诊疗推荐方案[J].中华儿科杂志,2006,44(8):596-598.
|
[21] |
Dinarello CA. Proinflammatory and anti-inflammatory cytokines as mediators in the pathogenesis of septic shock[J]. Chest, 1997, 112:S321-329.
|
[22] |
Lin TY, Chang LY, Huang YC, et al. Different proinflammatory reactions in fatal and non-fatal enterovirus 71 infections: Implications for early recognition and therapy[J]. Acta Paediatr, 2002, 91(6):632-635.
|
[23] |
Spittler A, Razenberger M, Kupper H, et al. Relationship between interleukin-6 plasma concentration in patients with sepsis, monocyte phenotype, monocyte phagocytic properties, and cytokine production[J].Clin Infect Dis, 2000, 31(6):1338-1420.
|
[24] |
Patel RT, Deen KI, Young SD, et al. Interleukin 6 is a prognostic indicator of outcome in severe intraabdominal sepsis[J]. Br J Surg, 1994, 81:1306-1308.
|
[25] |
Zhang F, Cao W. Tumor necrosis factor interleukin-6 and sepsis[J]. Surg Foreign Med Sci, 2005, 32(2):146-149.
|
[26] |
Yin W, Liang JH, Hu XM, et al. Significance of anisodamine on cytokine activity and the expression of cytokine mRNA in traumatic acute lung injury[J]. Chin J Exp Surg, 2000, 12:515-516.
|
[27] |
Zhang LD, Zhang XH. Changes of serum inflammatory cytokines and its significance in patients with multiple organ dysfunction syndrome[J]. J Intern Intensive Med, 2003, 9:38-39.
|
[28] |
Lin HY, Sheng ZY. Understanding change of systemic inflammatory response and MODS[J]. 2001, 13:643-646.
|
[29] |
Lin HW, Yue MX. Multiple organ dysfunction syndrome and immune imbalance[J]. Chin Crit Care Med, 2001, 13:565-567.
|
[30] |
Kawai S, Sakayori S, Kobayashi H. The role of IL-10 in patients with SIRS in relation to TNF activity[J]. Kansenshogaku Zasshi, 1995, 69:765-771.
|
[31] |
Ertel W, Keel M, Steckholzer U, et al. Interleukin-10 attenuates the releaseof proinflammatory cytokines but depresses splenocyte functions in murine endotoxemia[J]. Arch Surg, 1996, 131:51-56.
|
[32] |
Florquin S, Amyaoui Z, Goldman MJ, et al. Persistent production of TH2 type cytokines and polyclonal B cell activation after chronic administration of staphylococcal enterotoxin B in mice[J]. J Autoimmune, 1996, 9:609-615.
|
[33] |
van Dissel JT, Van Langevelde P, Westendorp RG, et al. Anti-inflammatory cytokine profile and mortality in febrile patients[J]. Lancet, 1998, 351:950-953.
|