[1] |
Lin QD, Qiu LH. Pathogenesis, diagnosis, and treatment of recurrent spontaneous abortion with immune type [J]. Front Med China, 2010, 4(3): 275-279.
|
[2] |
Hodes-Wertz B, Grifo J, Ghadir S, et al. Idiopathic recurrent miscarriage is caused mostly by aneuploid embryos [J]. Fertil Steril, 2012, 98(3): 675-680.
|
[3] |
Sugiura-Ogasawara M, Ozaki Y, Katano K, et al. Abnormal embryonic karyotype is the most frequent cause of recurrent miscarriage [J]. Hum Reprod, 2012, 27(8): 2297-2303.
|
[4] |
Abadie D, Hurault-Delarue C, Damase-Michel C, et al. Miscarriage [J]. J Am Med Assoc, 2015, 288(4):431-436.
|
[5] |
Chen H, Fu J, Huang W, et al. Dopamine agonists for preventing future miscarriage in women with idiopathic hyperprolactinemia and recurrent miscarriage history [J]. Cochrane Database Syst Rev, 2016, 7: CD008883.
|
[6] |
Mori M, Bogdan A, Balassa T, et al. The decidua-the maternal bed embracing the embryo-maintains the pregnancy [J]. Semin Immunopathol, 2016, 38(6): 635-649.
|
[7] |
Schatz F, Guzeloglu-Kayisli O, Arlier S, et al. The role of decidual cells in uterine hemostasis, menstruation, inflammation, adverse pregnancy outcomes and abnormal uterine bleeding [J]. Hum Reprod Update, 2016, 22(4): 497-515.
|
[8] |
Vinketova K, Mourdjeva M, Oreshkova T. Human decidual stromal cells as a component of the implantation niche and a modulator of maternal immunity [J]. J Pregnancy, 2016, 2016: 8689436.
|
[9] |
Clark DA, McDermott MR, Szewczuk MR. Impairment of host-versus-graft reaction in pregnant mice. Ⅱ. Selective suppression of cytotoxic T-cell generation correlates with soluble suppressor activity and with successful allogeneic pregnancy [J]. Cell Immunol, 1980, 52(1): 106-118.
|
[10] |
Qiu T, Teng Y, Wang Y, et al. Adoptive transfer of transforming growth factor-β1-induced CD4+CD25+ regulatory T cells prevents immune response-mediated spontaneous abortion [J]. 2015, 28(11): 1788-1797.
|
[11] |
Chen H, Song Y, Yang S, et al. YAP mediates human decidualization of the uterine endometrial stromal cells [J]. Placenta, 2017, 53: 30-35.
|
[12] |
Takeshita A, Kusakabe KT, Hiyama M, et al. Dynamics and reproductive effects of complement factors in the spontaneous abortion model of CBA/J×DBA/2 mice [J]. Immunobiology, 2014, 219(5): 385-391.
|
[13] |
Liu D, Huang Y, Chen B, et al. Activation of mammalian target of rapamycin pathway confers adverse outcome in nonsmall cell lung carcinoma [J]. Cancer, 117(16): 3763-3773.
|
[14] |
Clark DA, Chaouat G, Arck PC, et al. Cytokine-dependent abortion in CBA x DBA/2 mice is mediated by the procoagulant fgl2 prothrombinase [J]. J Immunol, 1998, 160(2): 545-549.
|
[15] |
Halder G, Johnson RL. Hippo signaling: growth control and beyond [J]. Development, 2011, 138(1): 9-22.
|
[16] |
Meng Z, Moroishi T, Guan KL. Mechanisms of Hippo pathway regulation [J]. Genes Dev, 2016, 30(1): 1-17.
|
[17] |
Lamar JM, Stern P, Liu H, et al. The Hippo pathway target, YAP, promotes metastasis through its TEAD-interaction domain [J]. Proc Natl Acad Sci U S A, 2012, 109(37): E2441-E2450.
|
[18] |
Santucci M, Vignudelli T, Ferrari S, et al. The Hippo pathway and YAP/TAZ-TEAD protein-protein interaction as targets for regenerative medicine and cancer treatment [J]. J Med Chem, 2015, 58(12): 4857-4873.
|